Thank you very much for the replies, gentlemen. The following is what I received from the Director of Hyperbaric Medicine via UHMS:
Quote:
I am the director of hyperbaric medicine at Virginia Mason Medical Center.
I am not aware of any table to correct for anemia.
I would be careful not to be hard and fast in using COHb levels to treat folks. Although it easy to measure, it is likely that most of the CO toxicity is not from the COHb but more due to direct CO toxicity. Levels often drop quickly due to pre-hospital O2 especially in a patient that is intubated. If someone has LOC or other significant severe symptoms we treat them even if their level is below 25%. Recall we are not treating to just get the level down but to help reduce the CO effects on other iron containing proteins as well as reducing inflammation and oxygenating ischemic tissue.
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Below are some articles that were forwarded to my email:
https://docs.google.com/viewer?a=v&p..._Gjyx2beXODUzg
https://docs.google.com/viewer?a=v&p...M-n4iYNLh0vC2w
https://docs.google.com/viewer?a=v&p...tM3Iq8GuOcjaSw
IMHO, I think Oxygen Content (CaO2) should be part of the evaluation criteria for HBOT.
Once levels get below 12vols%, weird things start happening at the cellular level.
In the numbers above, if a patients Hemoglobin is 7.3 gm/dL and their COHb is 14%, that would put the CaO2 at roughly a little over 8%. Considering the half life of COHb is 323 minutes, opposed to 23 minutes @ 3ATM, HBOT, IMO, would have been of benefit.
Thanks again.
Edited to add:
Below is what prompted the scenario:
http://newsfeed.time.com/2013/06/11/...rbon-monoxide/